Schizophrenia: Memory, Hallucinations, and Neurotransmissions Possible Correlations and Implications; Observational Analysis

1st Questions of interest:

Is it possible that abnormal deficits of the chemicals that excite neurons involved in memory cause abnormal surpluses that could have an adverse affect on other areas of the brain that are involved in processing auditory stimuli, and may alter perception?

2nd Question of interest:

Is it possible that auditory hallucinations experienced in people with schizophrenia may be a product of an over stimulation of auditory processes; thus, disabling these parts of the brain from perceiving selective stimuli, causing the stimuli to be intertwined, and in turn cause the person to hear abnormal sounds?

Behaviors in schizophrenic patients have been observed extensively. Many defects are distinguishable. Among them is memory, and recall insufficiencies. In addition, individuals who suffer from schizophrenia generally demonstrate disorganized thinking patterns, and may also experience delusions or hallucinations. The correlation between dopamine inadequacies and memory as well as recall deficiencies is comprehensible and widely supported. It is unclear, however, whether it is the only absolute determinant, or if it is just one factor among many. Studies have expressed that a potential factor may be a failure to organize the information to be encoded. Inferential data shows that the use of various stimulants is prevalent in people with schizophrenia. Further, people with schizophrenia seem to lack the ability to organize thoughts, or strategize. Perception and motivation seem to be absent in their life. In addition, a number of studies have shown that these individuals are moody, aggressive or homicidal in nature. Correlations have been discovered between cognition, recall, and memory. Further, false recognitions and other types of memory errors were positively associated with hallucinations. Experiments showed that antipsychotic medications used to treat Schizophrenia increased dopamine receptors in schizophrenic patients. Further, analysis indicated that increased blood flow is a major contributor to the effects of schizophrenia.